Martins-Oliveira, M., Akerman, S., Holland, P. R., Hoffmann, J. R., et al. (2017). Neuroendocrine signaling modulates specific neural networks relevant to migraine. Neurobiology of Disease, 101, 16 - 26.

Martins, I., & Tavares, I. (2017). Reticular formation and pain: The past and the future. Frontiers in Neuroanatomy, 11.

Martins, D., Silva, M., & Tavares, I. (2017). TRPV1 in pain control from the brain. Oncotarget, 8(10), 16101 - 16102.

Santos, S. S., Santos, S. M., Pinto, A. R. T., Ramu, V. G., et al. (2016). Amidated and ibuprofen-conjugated kyotorphins promote neuronal rescue and memory recovery in cerebral hypoperfusion dementia model. Frontiers in Aging Neuroscience, 8(JAN).

Lombo, C., Morgado, C., Tavares, I., & Neves, D. (2016). Effects of prolonged ingestion of epigallocatechin gallate on diabetes type 1-induced vascular modifications in the erectile tissue of rats. International Journal of Impotence Research, 28(4), 133 - 138.

Perazzo, J., Lopes-Ferreira, M., S. Santos, S., Serrano, I., et al. (2016). Endothelium-Mediated Action of Analogues of the Endogenous Neuropeptide Kyotorphin (Tyrosil-Arginine): Mechanistic Insights from Permeation and Effects on Microcirculation. ACS Chemical Neuroscience, 7(8), 1130 - 1140.

Silva, M., Martins, D., Charrua, A., Piscitelli, F., et al. (2016). Endovanilloid control of pain modulation by the rostroventromedial medulla in an animal model of diabetic neuropathy. Neuropharmacology, 107, 49 - 57.

Oliveira, M. C., Gano, L., Santos, I., Correia, J. D. G., et al. (2016). Improvement of the pharmacological properties of amidated kyotorphin by means of iodination. MedChemComm, 7(5), 906 - 913.

Oliveira, M. - M., Akerman, S., Tavares, I., & Goadsby, P. J. (2016). Neuropeptide y inhibits the trigeminovascular pathway through NPY Y1 receptor: Implications for migraine. Pain, 157(8), 1666 - 1673.

Silva, M., Costa-Pereira, J. T., Martins, D., & Tavares, I. (2016). Pain modulation from the brain during diabetic neuropathy: Uncovering the role of the rostroventromedial medulla. Neurobiology of Disease, 96, 346 - 356.

Serrano, I. D., Ramu, V. G., Pinto, A. R. T., Freire, J. M., et al. (2015). Correlation between membrane translocation and analgesic efficacy in kyotorphin derivatives. Biopolymers - Peptide Science Section, 104(1), 1 - 10.

Tavares, I. (2013). Human neural stem cell transplantation in spinal cord injury models: How far from clinical application?. Stem Cell Research and Therapy, 4(3).

Martins, I., De Vries, M. G., Teixeira-Pinto, A., Fadel, J., et al. (2013). Noradrenaline increases pain facilitation from the brain during inflammatory pain. Neuropharmacology, 71, 299 - 307.

Silva, M., Amorim, D., Almeida, A., Tavares, I., et al. (2013). Pronociceptive changes in the activity of rostroventromedial medulla (RVM) pain modulatory cells in the streptozotocin-diabetic rat. Brain Research Bulletin, 96, 39 - 44.

Ribeiro, M. M. B., Santos, S. S., Sousa, D. S. C., Oliveira, M., et al. (2013). Side-effects of analgesic kyotorphin derivatives: Advantages over clinical opioid drugs. Amino Acids, 45(1), 171 - 178.

Marques-Lopes, J., Martins, I., Pinho, D., Morato, M., et al. (2012). Decrease in the expression of N-methyl-D-aspartate receptors in the nucleus tractus solitarii induces antinociception and increases blood pressure. Journal of Neuroscience Research, 90(2), 356 - 366.

Morgado, C., Silva, L., Pereira-Terra, P., & Tavares, I. (2011). Changes in serotoninergic and noradrenergic descending pain pathways during painful diabetic neuropathy: The preventive action of IGF1. Neurobiology of Disease, 43(1), 275 - 284.

Ribeiro, M. M. B., Pinto, A. R. T., Domingues, M. M., Serrano, I., et al. (2011). Chemical conjugation of the neuropeptide kyotorphin and ibuprofen enhances brain targeting and analgesia. Molecular Pharmaceutics, 8(5), 1929 - 1940.

Pinho, D., Morato, M., Couto, M. R., Marques-Lopes, J., et al. (2011). Does chronic pain alter the normal interaction between cardiovascular and pain regulatory systems? Pain modulation in the hypertensive-monoarthritic rat. Journal of Pain, 12(2), 194 - 204.

Ribeiro, M. M. B., Pinto, A., Pinto, M., Heras, M., et al. (2011). Inhibition of nociceptive responses after systemic administration of amidated kyotorphin. British Journal of Pharmacology, 163(5), 964 - 973.

Morgado, C., Pereira-Terra, P., Cruz, C. D., & Tavares, I. (2011). Minocycline completely reverses mechanical hyperalgesia in diabetic rats through microglia-induced changes in the expression of the potassium chloride co-transporter 2 (KCC2) at the spinal cord. Diabetes, Obesity and Metabolism, 13(2), 150 - 159.

Martins, I., Cabral, L., Pinto, A., Wilson, S. P., et al. (2011). Reversal of inflammatory pain by HSV-1-mediated overexpression of enkephalin in the caudal ventrolateral medulla. European Journal of Pain, 15(10), 1008 - 1014.

Morgado, C., Pereira-Terra, P., & Tavares, I. (2011). ?-Lipoic acid normalizes nociceptive neuronal activity at the spinal cord of diabetic rats. Diabetes, Obesity and Metabolism, 13(8), 736 - 741.

Gonçalves, N., Rebelo, S., & Tavares, I. (2010). Fetal pain: Neurobiological causes and consequences. Dor fetal: Mecanismos neurobiológicos e consequênciasActa Medica Portuguesa, 23(3), 419 - 426.

Marques-Lopes, J., Pinho, D., Albino-Teixeira, A., & Tavares, I. (2010). The hyperalgesic effects induced by the injection of angiotensin II into the caudal ventrolateral medulla are mediated by the pontine A5 noradrenergic cell group. Brain Research, 1325, 41 - 52.

Morgado, C., Terra, P. P., & Tavares, I. (2010). Neuronal hyperactivity at the spinal cord and periaqueductal grey during painful diabetic neuropathy: Effects of gabapentin. European Journal of Pain, 14(7), 693 - 699.

Heinricher, M. M., Tavares, I., Leith, J. L., & Lumb, B. M. (2009). Descending control of nociception: Specificity, recruitment and plasticity. Brain Research Reviews, 60(1), 214 - 225.

Marques-Lopes, J., Pinto, M., Pinho, D., Morato, M., et al. (2009). Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia. Neuroscience, 158(4), 1301 - 1310.

Morgado, C., Pinto-Ribeiro, F., & Tavares, I. (2008). Diabetes affects the expression of GABA and potassium chloride cotransporter in the spinal cord: A study in streptozotocin diabetic rats. Neuroscience Letters, 438(1), 102 - 106.

Martins, I., Pinto, M., Wilson, S. P., Lima, D., & Tavares, I. (2008). Dynamic of migration of HSV-1 from a medullary pronociceptive centre: Antinociception by overexpression of the preproenkephalin transgene. European Journal of Neuroscience, 28(10), 2075 - 2083.

Pinto, M., Castro, A. R., Tshudy, F., Wilson, S. P., et al. (2008). Opioids modulate pain facilitation from the dorsal reticular nucleus. Molecular and Cellular Neuroscience, 39(4), 508 - 518.

Pinto, M., Sousa, M., Lima, D., & Tavares, I. (2008). Participation of ?-opioid, GABAB, and NK1 receptors of major pain control medullary areas in pathways targeting the rat spinal cord: Implications for descending modulation of nociceptive transmission. Journal of Comparative Neurology, 510(2), 175 - 187.

Morgado, C., & Tavares, I. (2007). C-fos expression at the spinal dorsal horn of streptozotocin-induced diabetic rats. Diabetes/Metabolism Research and Reviews, 23(8), 644 - 652.

Tavares, I., & Lima, D. (2007). From neuroanatomy to gene therapy: Searching for new ways to manipulate the supraspinal endogenous pain modulatory system. Journal of Anatomy, 211(2), 261 - 268.

Pinto, M., Lima, D., & Tavares, I. (2007). Neuronal activation at the spinal cord and medullary pain control centers after joint stimulation: A c-fos study in acute and chronic articular inflammation. Neuroscience, 147(4), 1076 - 1089.

Pinto, M., Lima, D., & Tavares, I. (2006). Correlation of noxious evoked c-fos expression in areas of the somatosensory system during chronic pain: Involvement of spino-medullary and intra-medullary connections. Neuroscience Letters, 409(2), 100 - 105.

Castro, A. R., Morgado, C., Lima, D., & Tavares, I. (2006). Differential expression of NK1 and GABAB receptors in spinal neurones projecting to antinociceptive or pronociceptive medullary centres. Brain Research Bulletin, 69(3), 266 - 275.

Morato, M., Pinho, D., Sousa, T., Tavares, I., & Albino-Teixeira, A. (2006). Inhibition of nociceptive responses of spinal cord neurones during hypertension involves the spinal GABAergic system and a pain modulatory center located at the caudal ventrolateral medulla. Journal of Neuroscience Research, 83(4), 647 - 655.

Pinho, D., Sousa, T., Morato, M., Tavares, I., & Albino-Teixeira, A. (2006). Lesion of the caudal ventrolateral medulla prevents the induction of hypertension by adenosine receptor blockade in rats. Brain Research, 1073-1074(1), 374 - 382.

Almeida, A., Leite-Almeida, H., & Tavares, I. (2006). Medullary control of nociceptive transmission: Reciprocal dual communication with the spinal cord. Drug Discovery Today: Disease Mechanisms, 3(3), 305 - 312.

Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2006). Secondary hyperalgesia in the monoarthritic rat is mediated by GABAB and NK1 receptors of spinal dorsal horn neurons: A behavior and c-fos study. Neuroscience, 141(4), 2087 - 2095.

Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2005). Imbalance between the expression of NK1 and GABAB receptors in nociceptive spinal neurons during secondary hyperalgesia: A c-Fos study in the monoarthritic rat. Neuroscience, 132(4), 905 - 916.

Tavares, I. (2004). The molecular metamorphosis of embryology: Implications for medical education. Medical Education, 38(5), 544.

Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2004). Nociceptive spinal neurons expressing NK1 and GABAB receptors are located in lamina I. Brain Research, 1003(1-2), 77 - 85.