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Author Title [ Type(Desc)] Year
Filters: First Letter Of Title is B and Author is Cruz, F.  [Clear All Filters]
Book Chapter
Avelino, A., Cruz, F., Panicker, J. N., & Liechti, M. D. (2016). Basic principles. In Practical Functional Urology (pp. 1 - 17).
Journal Article
Coelho, A., Antunes-Lopes, T., Gillespie, J., & Cruz, F. (2017). Beta-3 adrenergic receptor is expressed in acetylcholine-containing nerve fibers of the human urinary bladder: An immunohistochemical study. Neurourology and Urodynamics, 36(8), 1972 - 1980.
Antunes-Lopes, T., Carvalho-Barros, S., Cruz, C. - D., Cruz, F., & Martins-Silva, C. (2011). Biomarkers in overactive bladder: A new objective and noninvasive tool?. Advances in Urology.
Cruz, C. D., Coelho, A., Antunes-Lopes, T., & Cruz, F. (2015). Biomarkers of spinal cord injury and ensuing bladder dysfunction. Advanced Drug Delivery Reviews, 82, 153 - 159.
Dinis, P., Silva, J., Ribeiro, M. J., Avelino, A., et al. (2004). Bladder C-fiber desensitization induces a long-lasting improvement of BPH-associated storage LUTS: A pilot study. European Urology, 46(1), 88 - 94.
Laterza, R. M., Sievert, K. - D., de Ridder, D., Vierhout, M. E., et al. (2015). Bladder function after radical hysterectomy for cervical cancer. Neurourology and Urodynamics, 34(4), 309 - 315.
Matos, R., Cordeiro, J. M., Coelho, A., Ferreira, S., et al. (2016). Bladder pain induced by prolonged peripheral alpha 1A adrenoceptor stimulation involves the enhancement of transient receptor potential vanilloid 1 activity and an increase of urothelial adenosine triphosphate release. Acta Physiologica, 218(4), 265 - 275.
Silva, C., Silva, J., Castro, H., Reis, F., et al. (2007). Bladder sensory desensitization decreases urinary urgency. BMC Urology, 7.
Cruz, F., & Silva, C. (2004). Botulinum toxin in the management of lower urinary tract dysfunction: Contemporary update. Current Opinion in Urology, 14(6), 329 - 334.
Frias, B., Allen, S., Dawbarn, D., Charrua, A., et al. (2013). Brain-derived neurotrophic factor, acting at the spinal cord level, participates in bladder hyperactivity and referred pain during chronic bladder inflammation. Neuroscience, 234, 88 - 102.

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