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Author Title [ Type(Desc)] Year
Filters: Author is Gonçalves, N.P.  [Clear All Filters]
Journal Article
Lopes, C. D. F., Gonçalves, N. P., Gomes, C. P., Saraiva, M. J., & Pêgo, A. P. (2017). BDNF gene delivery mediated by neuron-targeted nanoparticles is neuroprotective in peripheral nerve injury. Biomaterials, 121, 83 - 96.
Ferreira, N., Gonçalves, N. P., Saraiva, M. J., & Almeida, M. R. (2016). Curcumin: A multi-Target disease-modifying agent for late-stage transthyretin amyloidosis. Scientific Reports, 6.
Gonçalves, N. P., Moreira, J., Martins, D., Vieira, P., et al. (2017). Differential expression of Cathepsin E in transthyretin amyloidosis: From neuropathology to the immune system. Journal of Neuroinflammation, 14(1).
Gonçalves, N. P., Martins, D., & Saraiva, M. J. (2017). The importance of pre-clinical studies in animal models of TTR amyloidosis for the discovery of novel patient disease biomarkers. Amyloid, 24, 83 - 84.
Martins, D., Moreira, J., Gonçalves, N. P., & Saraiva, M. J. (2017). MMP-14 overexpression correlates with the neurodegenerative process in familial amyloidotic polyneuropathy. DMM Disease Models and Mechanisms, 10(10), 1253 - 1260.
Gonçalves, N. P., Martins, D., & Saraiva, M. J. (2016). Overexpression of Protocadherin-10 in Transthyretin-Related Familial Amyloidotic Polyneuropathy. American Journal of Pathology, 186(7), 1913 - 1924.
Gonçalves, N. P., Gonçalves, P., Magalhaes, J., Ventosa, M., et al. (2016). Tissue remodeling after interference RNA mediated knockdown of transthyretin in a familial amyloidotic polyneuropathy mouse model. Neurobiology of Aging, 47, 91 - 101.

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