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Manning, A. L., Bakhoum, S. F., Maffini, S., Correia-Melo, C., et al. (2010). CLASP1, astrin and Kif2b form a molecular switch that regulates kinetochore-microtubule dynamics to promote mitotic progression and fidelity. EMBO Journal, 29(20), 3531 - 3543.
Logarinho, E., Maffini, S., Barisic, M., Marques, A., et al. (2012). CLASPs prevent irreversible multipolarity by ensuring spindle-pole resistance to traction forces during chromosome alignment. Nature Cell Biology, 14(3), 295 - 303.
Logarinho, E., Maffini, S., Barisic, M., Marques, A., et al. (2012). CLASPs prevent irreversible multipolarity by ensuring spindle-pole resistance to traction forces during chromosome alignment. Nature Cell Biology, 14(3), 295 - 303.
Logarinho, E., Maffini, S., Barisic, M., Marques, A., et al. (2012). CLASPs prevent irreversible multipolarity by ensuring spindle-pole resistance to traction forces during chromosome alignment. Nature Cell Biology, 14(3), 295 - 303.
Logarinho, E., Maffini, S., Barisic, M., Marques, A., et al. (2012). CLASPs prevent irreversible multipolarity by ensuring spindle-pole resistance to traction forces during chromosome alignment. Nature Cell Biology, 14(3), 295 - 303.
Barbosa, M., Sousa, A., Medeira, A., Lourenço, T., et al. (2011). Clinical and molecular characterization of Diastrophic Dysplasia in the Portuguese population. Clinical Genetics, 80(6), 550 - 557.
Barbosa, M., Sousa, A., Medeira, A., Lourenço, T., et al. (2011). Clinical and molecular characterization of Diastrophic Dysplasia in the Portuguese population. Clinical Genetics, 80(6), 550 - 557.
Dias, C. C., Rodrigues, P. P., da Costa-Pereira, A., & Magro, F. (2013). Clinical prognostic factors for disabling Crohn's disease: A systematic review and meta-analysis. World Journal of Gastroenterology, 19(24), 3866 - 3871.
Garriga, D., Vives-Adrián, L., Buxaderas, M., Ferreira-Da-Silva, F., et al. (2011). Cloning, purification and preliminary crystallographic studies of the 2AB protein from hepatitis A virus. Acta Crystallographica Section F: Structural Biology and Crystallization Communications, 67(10), 1224 - 1227.
Akhmanova, A., & Maiato, H. (2017). Closing the tubulin detyrosination cycle. Science, 358(6369), 1381 - 1382.
Magalhães, J., & Saraiva, M. J. (2011). Clusterin overexpression and its possible protective role in transthyretin deposition in familial amyloidotic polyneuropathy. Journal of Neuropathology and Experimental Neurology, 70(12), 1097 - 1106.
Pais-Vieira, M., Mendes-Pinto, M. M., Lima, D., & Galhardo, V. (2009). Cognitive impairment of prefrontal-dependent decision-making in rats after the onset of chronic pain. Neuroscience, 161(3), 671 - 679.
Pittman, A. M., Naranjo, S., Webb, E., Broderick, P., et al. (2009). The colorectal cancer risk at 18q21 is caused by a novel variant altering SMAD7 expression. Genome Research, 19(6), 987 - 993.
Clayton, G. M., Aller, S. G., Wang, J., Unger, V., & Morais-Cabral, J. H. (2009). Combining electron crystallography and X-ray crystallography to study the MlotiK1 cyclic nucleotide-regulated potassium channel. Journal of Structural Biology, 167(3), 220 - 226.
Ramos, E. M., Martins, S., Alonso, I., Emmel, V. E., et al. (2010). Common origin of pure and interrupted repeat expansions in spinocerebellar ataxia type 2 (SCA2). American Journal of Medical Genetics, Part B: Neuropsychiatric Genetics, 153(2), 524 - 531.
Ohmori, H., Ando, Y., Makita, Y., Onouchi, Y., et al. (2004). Common origin of the Val30Met mutation responsible for the amyloidogenic transthyretin type of familial amyloidotic polyneuropathy. Journal of medical genetics, 41(4).
Ohmori, H., Ando, Y., Makita, Y., Onouchi, Y., et al. (2004). Common origin of the Val30Met mutation responsible for the amyloidogenic transthyretin type of familial amyloidotic polyneuropathy. Journal of medical genetics, 41(4).
Lee, J. - M., Gillis, T., Mysore, J. S., Ramos, E. M., et al. (2012). Common SNP-based haplotype analysis of the 4p16.3 Huntington disease gene region. American Journal of Human Genetics, 90(3), 434 - 444.
Lee, J. - M., Gillis, T., Mysore, J. S., Ramos, E. M., et al. (2012). Common SNP-based haplotype analysis of the 4p16.3 Huntington disease gene region. American Journal of Human Genetics, 90(3), 434 - 444.
Lee, J. - M., Gillis, T., Mysore, J. S., Ramos, E. M., et al. (2012). Common SNP-based haplotype analysis of the 4p16.3 Huntington disease gene region. American Journal of Human Genetics, 90(3), 434 - 444.
Lee, J. - M., Gillis, T., Mysore, J. S., Ramos, E. M., et al. (2012). Common SNP-based haplotype analysis of the 4p16.3 Huntington disease gene region. American Journal of Human Genetics, 90(3), 434 - 444.
Lee, J. - M., Gillis, T., Mysore, J. S., Ramos, E. M., et al. (2012). Common SNP-based haplotype analysis of the 4p16.3 Huntington disease gene region. American Journal of Human Genetics, 90(3), 434 - 444.
Lee, J. - M., Gillis, T., Mysore, J. S., Ramos, E. M., et al. (2012). Common SNP-based haplotype analysis of the 4p16.3 Huntington disease gene region. American Journal of Human Genetics, 90(3), 434 - 444.
Lee, J. - M., Gillis, T., Mysore, J. S., Ramos, E. M., et al. (2012). Common SNP-based haplotype analysis of the 4p16.3 Huntington disease gene region. American Journal of Human Genetics, 90(3), 434 - 444.
Morales-Hojas, R., Vieira, C. P., Reis, M., & Vieira, J. (2009). Comparative analysis of five immunity-related genes reveals different levels of adaptive evolution in the virilis and melanogaster groups of Drosophila. Heredity, 102(6), 573 - 578.

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