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I
Mairal, T., Nieto, J., Pinto, M., Almeida, M. R., et al. (2009). Iodine atoms: a new molecular feature for the design of potent transthyretin fibrillogenesis inhibitors. PLoS ONE, 4(1).
Mairal, T., Nieto, J., Pinto, M., Almeida, M. R., et al. (2009). Iodine atoms: A new molecular feature for the design of potent transthyretin fibrillogenesis inhibitors. PLoS ONE, 4(1).
Gales, L., Almeida, M. R., Arsequell, G., Valencia, G., et al. (2008). Iodination of salicylic acid improves its binding to transthyretin. Biochimica et Biophysica Acta - Proteins and Proteomics, 1784(3), 512 - 517.
Gonçalves, N. P., Teixeira-Coelho, M., & Saraiva, M. J. (2014). The inflammatory response to sciatic nerve injury in a familial amyloidotic polyneuropathy mouse model. Experimental Neurology, 257, 76 - 87.
Monteiro, F. A., Cardoso, I., Sousa, M. M., & Saraiva, M. J. (2006). In vitro inhibition of transthyretin aggregate-induced cytotoxicity by full and peptide derived forms of the soluble receptor for advanced glycation end products (RAGE). FEBS Letters, 580(14), 3451 - 3456.
Gonçalves, N. P., Martins, D., & Saraiva, M. J. (2017). The importance of pre-clinical studies in animal models of TTR amyloidosis for the discovery of novel patient disease biomarkers. Amyloid, 24, 83 - 84.
Santos, S. D., Cardoso, I., Magalhães, J., & Saraiva, M. J. (2007). Impairment of the ubiquitin-proteasome system associated with extracellular transthyretin aggregates in familial amyloidotic polyneuropathy. Journal of Pathology, 213(2), 200 - 209.
Teixeira, C. A., Almeida, M. D. R., & Saraiva, M. J. (2016). Impairment of autophagy by TTR V30M aggregates: In vivo reversal by TUDCA and curcumin. Clinical Science, 130(18), 1665 - 1675.
Terazaki, H., Ando, Y., Fernandes, R., Yamamura, K. - I., et al. (2006). Immunization in familial amyloidotic polyneuropathy: Counteracting deposition by immunization with a Y78F TTR mutant. Laboratory Investigation, 86(1), 23 - 31.
H
Petrakis, I., Mavroeidi, V., Stylianou, K., Efthymiou, G., et al. (2013). Human TTRV30M localization within podocytes in a transgenic mouse model of transthyretin related amyloidosis: Does the environment play a role?. Transgenic Research, 22(1), 101 - 116.
Gales, L., Macedo-Ribeiro, S., Arsequell, G., Valencia, G., et al. (2005). Human transthyretin in complex with iododiflunisal: Structural features associated with a potent amyloid inhibitor. Biochemical Journal, 388(2), 615 - 621.
Martinho, A., Gonçalves, I., Cardoso, I., Almeida, M. R., et al. (2010). Human metallothioneins 2 and 3 differentially affect amyloid-beta binding by transthyretin. FEBS Journal, 277(16), 3427 - 3436.
Petrakis, I., Mavroeidi, V., Stylianou, K., Andronikidi, E., et al. (2013). Hsf-1 affects podocyte markers NPHS1, NPHS2 and WT1 in a transgenic mouse model of TTRVal30Met-related amyloidosis. Amyloid, 20(3), 164 - 172.
Batista, A. R., Sena-Esteves, M., & Saraiva, M. J. (2013). Hepatic production of transthyretin L12P leads to intracellular lysosomal aggregates in a new somatic transgenic mouse model. Biochimica et Biophysica Acta - Molecular Basis of Disease, 1832(8), 1183 - 1193.
Noborna, F., O'Callaghan, P., Hermansson, E., Zhang, X., et al. (2011). Heparan sulfate/heparin promotes transthyretin fibrillization through selective binding to a basic motif in the protein. Proceedings of the National Academy of Sciences of the United States of America, 108(14), 5584 - 5589.
Santos, S. D., Fernandes, R., & Saraiva, M. J. (2010). The heat shock response modulates transthyretin deposition in the peripheral and autonomic nervous systems. Neurobiology of Aging, 31(2), 280 - 289.
Magalhães, J., & Saraiva, M. J. (2012). The heat shock response in FAP: The role of the extracellular chaperone clusterin. Amyloid, 19(SUPPL. 1), 3 - 4.
Soares, M. L., Coelho, T., Sousa, A., Holmgren, G., et al. (2004). Haplotypes and DNA sequence variation within and surrounding the transthyretin gene: Genotype-phenotype correlations in familial amyloid polyneuropathy (V30M) in Portugal and Sweden. European Journal of Human Genetics, 12(3), 225 - 237.
G
Altland, K., Benson, M. D., Costello, C. E., Ferlini, A., et al. (2007). Genetic microheterogeneity of human transthyretin detected by IEF. Electrophoresis, 28(12), 2053 - 2064.
Oliveira, S. M., Ribeiro, C. A., Cardoso, I., & Saraiva, M. J. (2011). Gender-dependent transthyretin modulation of brain amyloid-β Levels: Evidence from a mouse model of alzheimer's disease. Journal of Alzheimer's Disease, 27(2), 429 - 439.
Plissonneau, M., Pansieri, J., Heinrich-Balard, L., Morfin, J. - F., et al. (2016). Gd-nanoparticles functionalization with specific peptides for ß-amyloid plaques targeting. Journal of Nanobiotechnology, 14(1).
F
Pires, R. H., Saraiva, M. J., Damas, A. M., & Kellermayer, M. S. Z. (2017). Force spectroscopy reveals the presence of structurally modified dimers in transthyretin amyloid annular oligomers. Journal of Molecular Recognition, 30(3).
Misumi, Y., Ando, Y., Gonçalves, N. P., & Saraiva, M. J. (2013). Fibroblasts endocytose and degrade transthyretin aggregates in transthyretin-related amyloidosis. Laboratory Investigation, 93(8), 911 - 920.
Saraiva, M. J., Sousa, M. M., Cardoso, I., & Fernandes, R. (2004). Familial amyloidotic polyneuropathy: Protein aggregation in the peripheral nervous system. Journal of Molecular Neuroscience, 23(1-2), 35 - 40.
E
Cardoso, I., Brito, M., & Saraiva, M. J. (2008). Extracellular matrix markers for disease progression and follow-up of therapies in familial amyloid polyneuropathy V30M TTR-related. Disease Markers, 25(1), 37 - 47.

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