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Costa, M. D. C., Teixeira-Castro, A., Constante, M., Magalhães, M., et al. (2006). Exclusion of mutations in the PRNP, JPH3, TBP, ATN1, CREBBP, POU3F2 and FTL genes as a cause of disease in Portuguese patients with a Huntington-like phenotype. Journal of Human Genetics, 51(8), 645 - 651.
Costa, M. D. C., Teixeira-Castro, A., Constante, M., Magalhães, M., et al. (2006). Exclusion of mutations in the PRNP, JPH3, TBP, ATN1, CREBBP, POU3F2 and FTL genes as a cause of disease in Portuguese patients with a Huntington-like phenotype. Journal of Human Genetics, 51(8), 645 - 651.
Aires, L., Silva, G., Martins, C., Marques, E., et al. (2016). Exercise intervention and cardiovascular risk factors in obese children. Comparison between obese youngsters taking part in a physical activity school-based programme with and without individualised diet counselling: the ACORDA project. Annals of Human Biology, 43(3), 183 - 190.
Aires, L., Silva, G., Martins, C., Marques, E., et al. (2016). Exercise intervention and cardiovascular risk factors in obese children. Comparison between obese youngsters taking part in a physical activity school-based programme with and without individualised diet counselling: the ACORDA project. Annals of Human Biology, 43(3), 183 - 190.
Aires, L., Silva, G., Martins, C., Marques, E., et al. (2016). Exercise intervention and cardiovascular risk factors in obese children. Comparison between obese youngsters taking part in a physical activity school-based programme with and without individualised diet counselling: the ACORDA project. Annals of Human Biology, 43(3), 183 - 190.
Seneca, S., Morris, M. A., Patton, S., Elles, R., & Sequeiros, J. (2008). Experience and outcome of 3 years of a European EQA scheme for genetic testing of the spinocerebellar ataxias. European Journal of Human Genetics, 16(8), 913 - 920.
Lopo, M., Montagud, A., Navarro, E., Cunha, I., et al. (2012). Experimental and modeling analysisof synechocystis sp. PCC 6803 growth. Journal of Molecular Microbiology and Biotechnology, 22(2), 71 - 82.
Lopo, M., Montagud, A., Navarro, E., Cunha, I., et al. (2012). Experimental and modeling analysisof synechocystis sp. PCC 6803 growth. Journal of Molecular Microbiology and Biotechnology, 22(2), 71 - 82.
Magalhães, A., Melo, P., Alves, C. J., Tavares, M. A., et al. (2008). Exploratory behavior in rats postnatally exposed to cocaine and housed in an enriched environment. Annals of the New York Academy of Sciences (Vol. 1139, pp. 358 - 365).
Magalhães, A., Melo, P., Alves, C. J., Tavares, M. A., et al. (2008). Exploratory behavior in rats postnatally exposed to cocaine and housed in an enriched environment. Annals of the New York Academy of Sciences (Vol. 1139, pp. 358 - 365).
Chavarria, D., Silva, T., Martins, D., Bravo, J., et al. (2015). Exploring cinnamic acid scaffold: development of promising neuroprotective lipophilic antioxidants. MedChemComm, 6(6), 1043 - 1053.
Mathieu-Daudé, F., Lafay, B., Touzet, O., Lelièvre, J., et al. (2008). Exploring the FL-160-CRP gene family through sequence variability of the complement regulatory protein (CRP) expressed by the trypomastigote stage of Trypanosoma cruzi. Infection, Genetics and Evolution, 8(3), 258 - 266.
Oliveira, A. V., Vilaça, R., Santos, C. N., Costa, V., & Menezes, R. (2017). Exploring the power of yeast to model aging and age-related neurodegenerative disorders. Biogerontology, 18(1), 3 - 34.
Pereira, L. G., Coimbra, S., Oliveira, H., Monteiro, L., & Sottomayor, M. (2006). Expression of arabinogalactan protein genes in pollen tubes of Arabidopsis thaliana. Planta, 223(2), 374 - 380.
Campos, S. P., de Lurdes Pinto, M., Gomes, G., de Pinho, P. G., et al. (2016). Expression of CYP1A1 and CYP1A2 in the liver and kidney of rabbits after prolonged infusion of propofol. Experimental and Toxicologic Pathology, 68(9), 521 - 531.
Marques, O., Canadas, A., Faria, F., Oliveira, E., et al. (2017). Expression of iron-related proteins in feline and canine mammary gland reveals unexpected accumulation of iron. Biotechnic and Histochemistry.
Braz, S. O., Cruz, A., Lobo, A., Bravo, J., et al. (2017). Expression of Rac1 alternative 3′ UTRs is a cell specific mechanism with a function in dendrite outgrowth in cortical neurons. Biochimica et Biophysica Acta - Gene Regulatory Mechanisms, 1860(6), 685 - 694.
Braz, S. O., Cruz, A., Lobo, A., Bravo, J., et al. (2017). Expression of Rac1 alternative 3′ UTRs is a cell specific mechanism with a function in dendrite outgrowth in cortical neurons. Biochimica et Biophysica Acta - Gene Regulatory Mechanisms, 1860(6), 685 - 694.
Castro, M. A. A., Oliveira, M. I., Nunes, R. J., Fabre, S., et al. (2007). Extracellular isoforms of CD6 generated by alternative splicing regulate targeting of CD6 to the immunological synapse. Journal of Immunology, 178(7), 4351 - 4361.
Borges, G. S., Berrocoso, E., Ortega-Alvaro, A., Mico, J. A., & Neto, F. L. (2013). Extracellular signal-regulated kinase activation in the chronic constriction injury model of neuropathic pain in anaesthetized rats. European Journal of Pain (United Kingdom), 17(1), 35 - 45.
F
Horta, R., Aguiar, P., Monteiro, D., Silva, A., & Amarante, J. M. (2014). A facegram for spatial-temporal analysis of facial excursion: Applicability in the microsurgical reanimation of long-standing paralysis and pretransplantation. Journal of Cranio-Maxillofacial Surgery.
Horta, R., Aguiar, P., Silva, P., Monteiro, D., & Amarante, J. M. (2014). Facial disfigurement: Restoration of facial dynamics in a patient with concomitant facial paralysis and blindness. Journal of Reconstructive Microsurgery, 30(3), 207 - 210.
Pinho, T., MacIel, P., Lemos, C., & Sousa, A. (2010). Familial aggregation of maxillary lateral incisor agenesis. Journal of Dental Research, 89(6), 621 - 625.
Munar-Qués, M., Masjuan, J., Coelho, T., Moreira, P., et al. (2007). Familial amyloid polyneuropathy associated with TTRSer50Arg mutation in two Iberian families presenting a novel single base change in the mutant gene. Amyloid, 14(2), 147 - 152.
Munar-Qués, M., Masjuan, J., Coelho, T., Moreira, P., et al. (2007). Familial amyloid polyneuropathy associated with TTRSer50Arg mutation in two Iberian families presenting a novel single base change in the mutant gene. Amyloid, 14(2), 147 - 152.

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