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Ascensão, A., Magalhães, J., Soares, J., Ferreira, R., et al. (2005). Endurance training attenuates doxorubicin-induced cardiac oxidative damage in mice. International Journal of Cardiology, 100(3), 451 - 460.
Ascensão, A., Magalhães, J., Soares, J. M. C., Ferreira, R., et al. (2006). Endurance training limits the functional alterations of heart rat mitochondria submitted to in vitro anoxia-reoxygenation. International Journal of Cardiology, 109(2), 169 - 178.
Gomes, J. R. B., Sousa, E. A., Gonçalves, J. M., Gales, L., et al. (2007). Energetic and structural characterization of 2-R-3-methylquinoxaline-1,4- dioxides (R = benzoyl or tert-butoxycarbonyl): Experimental and computational studies. Journal of Physical Organic Chemistry, 20(7), 491 - 498.
Tomada, I., Fernandes, D., Guimarães, J. T., Almeida, H., & Neves, D. (2012). Energy restriction ameliorates metabolic syndrome-induced cavernous tissue structural modifications in aged rats. Age.
Tomada, I., Tomada, N., Almeida, H., & Neves, D. (2012). Energy restriction and exercise modulate angiopoietins and vascular endothelial growth factor expression in the cavernous tissue of high-fat diet-fed rats. Asian Journal of Andrology, 14(4), 635 - 642.
Salomé Gomes, M., Fernandes, S. S., Cordeiro, J. V., Gomes, S. S., et al. (2008). Engagement of Toll-like receptor 2 in mouse macrophages infected with Mycobacterium avium induces non-oxidative and TNF-independent anti-mycobacterial activity. European Journal of Immunology, 38(8), 2180 - 2189.
Ramos, P., Guy, E., Chen, N., Proenca, C. C., et al. (2011). Enhanced erythropoiesis in Hfe-KO mice indicates a role for Hfe in the modulation of erythroid iron homeostasis. Blood, 117(4), 1379 - 1389.
Harley, C. A., Starek, G., Jones, D. K., Fernandes, A. S., et al. (2016). Enhancement of hERG channel activity by scFv antibody fragments targeted to the PAS domain. Proceedings of the National Academy of Sciences of the United States of America, 113(35), 9916 - 9921.
Morais-Cabral, J. H., & Robertson, G. A. (2015). The enigmatic cytoplasmic regions of KCNH channels. Journal of Molecular Biology, 427(1), 67 - 76.
Santos, S. D., & Saraiva, M. J. (2004). Enlarged ventricles, astrogliosis and neurodegeneration in heat shock factor 1 null mouse brain. Neuroscience, 126(3), 657 - 663.
Barata, L., Sousa Silva, M., Schuldt, L., Ferreira, A. E. N., et al. (2011). Enlightening the molecular basis of trypanothione specificity in trypanosomatids: Mutagenesis of Leishmania infantum glyoxalase II. Experimental Parasitology, 129(4), 402 - 408.
Veiga-Malta, I., Duarte, M., Dinis, M., Tavares, D., et al. (2004). Enolase from Streptococcus sobrinus is an immunosuppressive protein. Cellular Microbiology, 6(1), 79 - 88.
Teixeira, L., Marques, R. M., Ferreirinha, P., Bezerra, F., et al. (2016). Enrichment of IFN-γ producing cells in different murine adipose tissue depots upon infection with an apicomplexan parasite. Scientific Reports, 6.
Olsson, I. A. S., Costa, A., Nobrega, C., Roque, S., & Correia-Neves, M. (2010). Environmental enrichment does not compromise the immune response in mice chronically infected with mycobacterium avium. Scandinavian Journal of Immunology, 71(4), 249 - 257.
Harmatz, P., Giugliani, R., Schwartz, I., Guffon, N., et al. (2006). Enzyme replacement therapy for mucopolysaccharidosis VI: A phase 3, randomized, double-blind, placebo-controlled, multinational study of recombinant human N-acetylgalactosamine 4-sulfatase (recombinant human arylsulfatase B or rhASB) and follow-on, open-l. Journal of Pediatrics, 148(4), 533 - 533.e.
Harmatz, P., Yu, Z. - F., Giugliani, R., Schwartz, I. V. D., et al. (2010). Enzyme replacement therapy for mucopolysaccharidosis VI: Evaluation of long-term pulmonary function in patients treated with recombinant human N-acetylgalactosamine 4-sulfatase. Journal of Inherited Metabolic Disease, 33(1), 51 - 60.
Decker, C., Yu, Z. - F., Giugliani, R., Schwartz, I. V. D., et al. (2010). Enzyme replacement therapy for mucopolysaccharidosis VI: Growth and pubertal development in patients treated with recombinant human N-acetylgalactosamine 4-sulfatase. Journal of Pediatric Rehabilitation Medicine, 3(2), 89 - 100.
Braunlin, E., Rosenfeld, H., Kampmann, C., Johnson, J., et al. (2013). Enzyme replacement therapy for mucopolysaccharidosis VI: Long-term cardiac effects of galsulfase (Naglazyme®) therapy. Journal of Inherited Metabolic Disease, 36(2), 385 - 394.
Braunlin, E., Rosenfeld, H., Kampmann, C., Johnson, J., et al. (2012). Enzyme replacement therapy for mucopolysaccharidosis VI: long-term cardiac effects of galsulfase (Naglazyme ®) therapy. Journal of Inherited Metabolic Disease.
MacEdo, M. F., Quinta, R., Pereira, C. S., & Sa Miranda, M. C. (2012). Enzyme replacement therapy partially prevents invariant Natural Killer T cell deficiency in the Fabry disease mouse model. Molecular Genetics and Metabolism, 106(1), 83 - 91.
Sequeiros, J., Martins, S., & Silveira, I. (2011). Epidemiology and population genetics of degenerative ataxias. Handbook of Clinical Neurology (Vol. 103, pp. 227 - 251).
Azevedo, L. F., Costa-Pereira, A., Mendonça, L., Dias, C. C., & Castro-Lopes, J. M. (2012). Epidemiology of chronic pain: A population-based nationwide study on its prevalence, characteristics and associated disability in Portugal. Journal of Pain, 13(8), 773 - 783.
Ferreira, N., Saraiva, M. J., & Almeida, M. R. (2012). Epigallocatechin-3-gallate as a potential therapeutic drug for TTR-related amyloidosis: "In vivo" evidence from FAP mice models. PLoS ONE, 7(1).
Oikemus, S. R., Queiroz-Machado, J., Lai, K., McGinnis, N., et al. (2006). Epigenetic telomere protection by Drosophila DNA damage response pathways. PLoS Genetics, 2(5), 693 - 706.
Oikemus, S. R., Queiroz-Machado, J., Lai, K., McGinnis, N., et al. (2006). Epigenetic telomere protection by Drosophila DNA damage response pathways. PLoS genetics, 2(5).

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