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Belinha, I., Amorim, M. A., Rodrigues, P., de Freitas, V., et al. (2007). Quercetin increases oxidative stress resistance and longevity in Saccharomyces cerevisiae. Journal of Agricultural and Food Chemistry, 55(6), 2446 - 2451.
Belo, L., Santos-Silva, A., Rocha, S., Caslake, M., et al. (2005). Fluctuations in C-reactive protein concentration and neutrophil activation during normal human pregnancy. European Journal of Obstetrics Gynecology and Reproductive Biology, 123(1), 46 - 51.
Belo, L., Santos-Silva, A., Quintanilha, A., & Rebelo, I. (2006). Pre-eclampsia versus cardiovascular disease versus CRP. Current Hypertension Reviews, 2(4), 317 - 323.
Belo, L., Santos-Silva, A., Lowe, G., Rumley, A., et al. (2005). High-density lipoprotein particles may regulate hemostasis in human pregnancy. Fertility and Sterility, 84(4), 1021 - 1022.
Belo, L., Santos-Silva, A., Caslake, M., Pereira-Leite, L., et al. (2005). Oxidized-LDL levels in normal and pre-eclamptic pregnancies: Contribution of LDL particle size [2]. Atherosclerosis, 183(1), 185 - 186.
Belo, L., Santos-Silva, A., Quintanilha, A., & Rebelo, I. (2008). Similarities between pre-eclampsia and atherosclerosis: A protective effect of physical exercise?. Current Medicinal Chemistry, 15(22), 2223 - 2229.
Belo, R., Santarém, N., Pereira, C., Pérez-Cabezas, B., et al. (2017). Leishmania infantum exoproducts inhibit human invariant NKT cell expansion and activation. Frontiers in Immunology, 8(JUN).
Belo, L., Caslake, M., Santos-Silva, A., Castro, E. M. B., et al. (2004). LDL size, total antioxidant status and oxidised LDL in normal human pregnancy: A longitudinal study. Atherosclerosis, 177(2), 391 - 399.
Belo, L., Gaffney, D., Caslake, M., Santos-Silva, A., et al. (2004). Apolipoprotein e and cholesteryl ester transfer protein polymorphisms in normal and preeclamptic pregnancies. European Journal of Obstetrics Gynecology and Reproductive Biology, 112(1), 9 - 15.
Benninger, Y., Thurnherr, T., Pereira, J. A., Krause, S., et al. (2007). Essential and distinct roles for cdc42 and rac1 in the regulation of Schwann cell biology during peripheral nervous system development. Journal of Cell Biology, 177(6), 1051 - 1061.
Bento, V. F., Cruz, V. T., Ribeiro, D. D., Branco, C., & Coutinho, P. (2013). The potential of motion quantification systems in the automatic evaluation of motor function after stroke. International Journal of Stroke, 8(6), - .
Berce, V., Kozmus, C. E. P., & Potočnik, U. (2016). CTLA4 expression in childhood asthma and the effect of treatment with inhaled corticosteroid and Leukotriene receptor antagonist. Annual Research and Review in Biology, 10(2).
Berce, V., Kozmus, C. E. P., & Potočnik, U. (2012). Association among ORMDL3 gene expression, 17q21 polymorphism and response to treatment with inhaled corticosteroids in children with asthma. Pharmacogenomics Journal.
Bessa, C., Pereira, C., Leão, M., Maciel, C., et al. (2013). Using yeast to uncover the regulation of protein kinase Cδ by ceramide. FEMS Yeast Research, 13(7), 700 - 705.
Bessa, J., Tavares, M. J., Santos, J., Kikuta, H., et al. (2008). meis1 regulates cyclin D1 and c-myc expression, and controls the proliferation of the multipotent cells in the early developing zebrafish eye. Development, 135(5), 799 - 803.
Bessa, J., & Casares, F. (2005). Restricted teashirt expression confers eye-specific responsiveness to Dpp and Wg signals during eye specification in Drosophila. Development, 132(22), 5011 - 5020.
Bessa, J., Tena, J. J., De La Calle-Mustienes, E., Fernández-Miñán, A., et al. (2009). Zebrafish Enhancer Detection (ZED) vector: A new tool to facilitate transgenesis and the functional analysis of cis-regulatory regions in zebrafish. Developmental Dynamics, 238(9), 2409 - 2417.
Bessa, C., Teixeira, C. A. F., Mangas, M., Dias, A., et al. (2006). Two novel CLN5 mutations in a Portuguese patient with vLINCL: Insights into molecular mechanisms of CLN5 deficiency. Molecular Genetics and Metabolism, 89(3), 245 - 253.
Bessa, J., Carmona, L., & Casares, F. (2009). Zinc-finger paralogues tsh and tio are functionally equivalent during imaginal development in Drosophila and maintain their expression levels through auto- and cross-negative feedback loops. Developmental Dynamics, 238(1), 19 - 28.
Bettencourt, C., Raposo, M., Kazachkova, N., Cymbron, T., et al. (2011). The APOE ε 2 allele increases the risk of earlier age at onset in Machado-Joseph disease. Archives of Neurology, 68(12), 1580 - 1583.
Bettencourt, C., Raposo, M., Kazachkova, N., Santos, C., et al. (2012). Sequence analysis of 5′ regulatory regions of the Machado-Joseph disease gene (ATXN3). Cerebellum, 11(4), 1045 - 1050.
Bettencourt, C., Quintáns, B., Ros, R., Ampuero, I., et al. (2012). Revisiting genotype-phenotype overlap in neurogenetics: Triplet-repeat expansions mimicking spastic paraplegias. Human Mutation, 33(9), 1315 - 1323.
Bettencourt, C., Santos, C., Coutinho, P., Rizzu, P., et al. (2011). Parkinsonian phenotype in Machado-Joseph disease (MJD/SCA3): A two-case report. BMC Neurology, 11.
Bettencourt, C., Fialho, R. N., Santos, C., Montiel, R., et al. (2008). Segregation distortion of wild-type alleles at the Machado-Joseph disease locus: A study in normal families from the Azores islands (Portugal). Journal of Human Genetics, 53(4), 333 - 339.
Bettencourt, C., Raposo, M., Ros, R., Montiel, R., et al. (2012). Transcript Diversity of Machado-Joseph Disease Gene (ATXN3) Is Not Directly Determined by SNPs in Exonic or Flanking Intronic Regions. Journal of Molecular Neuroscience.


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