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Author Title [ Type(Desc)] Year
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Journal Article
Sant'Anna, R., Almeida, M. R., Varejao, N., Gallego, P., et al. (2017). Cavity filling mutations at the thyroxine-binding site dramatically increase transthyretin stability and prevent its aggregation. Scientific Reports, 7.
Teixeira, L., Marques, A., Meireles, C. S., Seabra, A. R., et al. (2005). Characterization of the B-cell immune response elicited in BALB/c mice challenged with Neospora caninum tachyzoites. Immunology, 116(1), 38 - 52.
Pereira, P. S., Teixeira, A., Pinho, S., Ferreira, P., et al. (2006). E-cadherin missense mutations, associated with hereditary diffuse gastric cancer (HDGC) syndrome, display distinct invasive behaviors and genetic interactions with the Wnt and Notch pathways in Drosophila epithelia. Human Molecular Genetics, 15(10), 1704 - 1712.
Dinis, M., Trigo, G., Chaves, N., Fonseca, A. J. M. M., et al. (2011). eEnolase maternal immunization confers caries protection on offspring. Journal of Dental Research, 90(3), 325 - 330.
Veiga-Malta, I., Duarte, M., Dinis, M., Tavares, D., et al. (2004). Enolase from Streptococcus sobrinus is an immunosuppressive protein. Cellular Microbiology, 6(1), 79 - 88.
Oliveira, L., Madureira, P., Andrade, E. B., Bouaboud, A., et al. (2012). Group B streptococcus GAPDH is released upon cell lysis, associates with bacterial surface, and induces apoptosis in murine macrophages. PLoS ONE, 7(1).
Magalhães, V., Andrade, E. B., Alves, J., Ribeiro, A., et al. (2013). Group B Streptococcus Hijacks the Host Plasminogen System to Promote Brain Endothelial Cell Invasion. PLoS ONE, 8(5).
Trigo, G., Ferreira, P., Ribeiro, N., Dinis, M., et al. (2008). Identification of immunoreactive extracellular proteins of Streptococcus agalactiae in bovine mastitis. Canadian Journal of Microbiology, 54(11), 899 - 905.
Veiga-Malta, I., Duarte, M., Dinis, M., Madureira, P., et al. (2004). Identification of NAD+ Synthetase from Streptococcus sobrinus as a B-Cell-Stimulatory Protein. Journal of Bacteriology, 186(2), 419 - 426.
Camejo, A., Buchrieser, C., Couvé, E., Carvalho, F., et al. (2009). In vivo transcriptional profiling of Listeria monocytogenes and mutagenesis identify new virulence factors involved in infection. PLoS Pathogens, 5(5).
Madureira, P., Andrade, E. B., Gama, B., Oliveira, L., et al. (2011). Inhibition of IL-10 production by maternal antibodies against group b streptococcus GAPDH confers immunity to offspring by favoring neutrophil recruitment. PLoS Pathogens, 7(11).
Magalhães, V., Veiga-Malta, I., Almeida, M. R., Baptista, M., et al. (2007). Interaction with human plasminogen system turns on proteolytic activity in Streptococcus agalactiae and enhances its virulence in a mouse model. Microbes and Infection, 9(11), 1276 - 1284.
Trigo, G., Dinis, M., França, Â., Andrade, E. B., et al. (2009). Leukocyte populations and cytokine expression in the mammary gland in a mouse model of Streptococcus agalactiae mastitis. Journal of Medical Microbiology, 58(7), 951 - 958.
Dinis, M., Tavares, D., Veiga-Malta, I., Fonseca, A. J. M. M., et al. (2009). Oral therapeutic vaccination with Streptococcus sobrinus recombinant enolase confers protection against dental caries in rats. Journal of Infectious Diseases, 199(1), 116 - 123.
Vilanova, M., Teixeira, L., Caramalho, Í., Torrado, E., et al. (2004). Protection against systemic candidiasis in mice immunized with secreted aspartic proteinase 2. Immunology, 111(3), 334 - 342.
Barradas, P. F., Vilhena, H., Oliveira, A. C., Granada, S., et al. (2017). Serological and molecular detection of spotted fever group Rickettsia in a group of pet dogs from Luanda, Angola. Parasites and Vectors, 10(1).
Madureira, P., Baptista, M., Vieira, M., Magalhães, V., et al. (2007). Streptococcus agalactiae GAPDH is a virulence-associated immunomodulatory protein. Journal of Immunology, 178(3), 1379 - 1387.
Dinis, M., Tavares, D., Fonseca, A. J. M. M., Faria, R., et al. (2004). Therapeutic vaccine against Streptococcus sobrinus-induced caries. Journal of Dental Research, 83(4), 354 - 358.
Andrade, E. B., Alves, J., Madureira, P., Oliveira, L., et al. (2013). TLR2-induced IL-10 production impairs neutrophil recruitment to infected tissues during neonatal bacterial sepsis. Journal of Immunology, 191(9), 4759 - 4768.

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