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Ribeiro, C. A., Saraiva, M. J., & Cardoso, I. (2012). Stability of the Transthyretin Molecule as a Key Factor in the Interaction with A-Beta Peptide - Relevance in Alzheimer's Disease. PLoS ONE, 7(9).
Cardoso, I., Martins, D., Ribeiro, T., Merlini, G., & Saraiva, M. J. (2010). Synergy of combined Doxycycline/TUDCA treatment in lowering Transthyretin deposition and associated biomarkers: Studies in FAP mouse models. Journal of Translational Medicine, 8.
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Conejos-Sánchez, I., Cardoso, I., Saraiva, M. J., & Vicent, M. J. (2014). Targeting a rare amyloidotic disease through rationally designed polymer conjugates. Journal of Controlled Release, 178(1), 95 - 100.
Costa, R., Speretta, E., Crowther, D. C., & Cardoso, I. (2011). Testing the therapeutic potential of doxycycline in a Drosophila melanogaster model of Alzheimer disease. Journal of Biological Chemistry, 286(48), 41647 - 41655.
Sousa, J. C., Cardoso, I., Marques, F., Saraiva, M. J., & Palha, J. A. (2007). Transthyretin and Alzheimer's disease: Where in the brain?. Neurobiology of Aging, 28(5), 713 - 718.
Costa, R., Gonçalves, A., Saraiva, M. J., & Cardoso, I. (2008). Transthyretin binding to A-Beta peptide - Impact on A-Beta fibrillogenesis and toxicity. FEBS Letters, 582(6), 936 - 942.
Ribeiro, C. A., Santana, I., Oliveira, C., Baldeiras, I., et al. (2012). Transthyretin decrease in plasma of MCI and AD patients: Investigation of mechanisms for disease modulation. Current Alzheimer Research, 9(8), 881 - 889.
Silva, C. S., Eira, J., Ribeiro, C. A., Oliveira, Â., et al. (2017). Transthyretin neuroprotection in Alzheimer's disease is dependent on proteolysis. Neurobiology of Aging, 59, 10 - 14.
Alemi, M., Gaiteiro, C., Ribeiro, C. A., Santos, L. M., et al. (2016). Transthyretin participates in beta-amyloid transport from the brain to the liver- involvement of the low-density lipoprotein receptor-related protein 1?. Scientific Reports, 6.
Costa, R., Ferreira-Da-Silva, F., Saraiva, M. J., & Cardoso, I. (2008). Transthyretin protects against A-beta peptide toxicity by proteolytic cleavage of the peptide: A mechanism sensitive to the kunitz protease inhibitor. PLoS ONE, 3(8).
Alemi, M., Silva, S. C., Santana, I., & Cardoso, I. (2017). Transthyretin stability is critical in assisting beta amyloid clearance– Relevance of transthyretin stabilization in Alzheimer's disease. CNS Neuroscience and Therapeutics, 23(7), 605 - 619.
Ribeiro, C. A., Oliveira, S. M., Guido, L. F., Magalhães, A., et al. (2014). Transthyretin stabilization by iododiflunisal promotes amyloid-β peptide clearance, decreases its deposition, and ameliorates cognitive deficits in an Alzheimer's disease mouse model. Journal of Alzheimer's Disease, 39(2), 357 - 370.

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