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Α
Morgado, C., Pereira-Terra, P., & Tavares, I. (2011). α-Lipoic acid normalizes nociceptive neuronal activity at the spinal cord of diabetic rats. Diabetes, Obesity and Metabolism, 13(8), 736 - 741.
T
Martins, D., Silva, M., & Tavares, I. (2017). TRPV1 in pain control from the brain. Oncotarget, 8(10), 16101 - 16102.
S
Ribeiro, M. M. B., Santos, S. S., Sousa, D. S. C., Oliveira, M., et al. (2013). Side-effects of analgesic kyotorphin derivatives: Advantages over clinical opioid drugs. Amino Acids, 45(1), 171 - 178.
Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2006). Secondary hyperalgesia in the monoarthritic rat is mediated by GABAB and NK1 receptors of spinal dorsal horn neurons: A behavior and c-fos study. Neuroscience, 141(4), 2087 - 2095.
R
Martins, I., Cabral, L., Pinto, A., Wilson, S. P., et al. (2011). Reversal of inflammatory pain by HSV-1-mediated overexpression of enkephalin in the caudal ventrolateral medulla. European Journal of Pain, 15(10), 1008 - 1014.
Martins, I., & Tavares, I. (2017). Reticular formation and pain: The past and the future. Frontiers in Neuroanatomy, 11.
P
Silva, M., Amorim, D., Almeida, A., Tavares, I., et al. (2013). Pronociceptive changes in the activity of rostroventromedial medulla (RVM) pain modulatory cells in the streptozotocin-diabetic rat. Brain Research Bulletin, 96, 39 - 44.
Pinto, M., Sousa, M., Lima, D., & Tavares, I. (2008). Participation of μ-opioid, GABAB, and NK1 receptors of major pain control medullary areas in pathways targeting the rat spinal cord: Implications for descending modulation of nociceptive transmission. Journal of Comparative Neurology, 510(2), 175 - 187.
Silva, M., Costa-Pereira, J. T., Martins, D., & Tavares, I. (2016). Pain modulation from the brain during diabetic neuropathy: Uncovering the role of the rostroventromedial medulla. Neurobiology of Disease, 96, 346 - 356.
O
Pinto, M., Castro, A. R., Tshudy, F., Wilson, S. P., et al. (2008). Opioids modulate pain facilitation from the dorsal reticular nucleus. Molecular and Cellular Neuroscience, 39(4), 508 - 518.
N
Martins, I., De Vries, M. G., Teixeira-Pinto, A., Fadel, J., et al. (2013). Noradrenaline increases pain facilitation from the brain during inflammatory pain. Neuropharmacology, 71, 299 - 307.
Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2004). Nociceptive spinal neurons expressing NK1 and GABAB receptors are located in lamina I. Brain Research, 1003(1-2), 77 - 85.
Oliveira, M. - M., Akerman, S., Tavares, I., & Goadsby, P. J. (2016). Neuropeptide y inhibits the trigeminovascular pathway through NPY Y1 receptor: Implications for migraine. Pain, 157(8), 1666 - 1673.
Morgado, C., Terra, P. P., & Tavares, I. (2010). Neuronal hyperactivity at the spinal cord and periaqueductal grey during painful diabetic neuropathy: Effects of gabapentin. European Journal of Pain, 14(7), 693 - 699.
Pinto, M., Lima, D., & Tavares, I. (2007). Neuronal activation at the spinal cord and medullary pain control centers after joint stimulation: A c-fos study in acute and chronic articular inflammation. Neuroscience, 147(4), 1076 - 1089.
Martins-Oliveira, M., Akerman, S., Holland, P. R., Hoffmann, J. R., et al. (2017). Neuroendocrine signaling modulates specific neural networks relevant to migraine. Neurobiology of Disease, 101, 16 - 26.
M
Tavares, I. (2004). The molecular metamorphosis of embryology: Implications for medical education. Medical Education, 38(5), 544.
Morgado, C., Pereira-Terra, P., Cruz, C. D., & Tavares, I. (2011). Minocycline completely reverses mechanical hyperalgesia in diabetic rats through microglia-induced changes in the expression of the potassium chloride co-transporter 2 (KCC2) at the spinal cord. Diabetes, Obesity and Metabolism, 13(2), 150 - 159.
Marques-Lopes, J., Pinto, M., Pinho, D., Morato, M., et al. (2009). Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia. Neuroscience, 158(4), 1301 - 1310.
Almeida, A., Leite-Almeida, H., & Tavares, I. (2006). Medullary control of nociceptive transmission: Reciprocal dual communication with the spinal cord. Drug Discovery Today: Disease Mechanisms, 3(3), 305 - 312.
L
Pinho, D., Sousa, T., Morato, M., Tavares, I., & Albino-Teixeira, A. (2006). Lesion of the caudal ventrolateral medulla prevents the induction of hypertension by adenosine receptor blockade in rats. Brain Research, 1073-1074(1), 374 - 382.
I
Morato, M., Pinho, D., Sousa, T., Tavares, I., & Albino-Teixeira, A. (2006). Inhibition of nociceptive responses of spinal cord neurones during hypertension involves the spinal GABAergic system and a pain modulatory center located at the caudal ventrolateral medulla. Journal of Neuroscience Research, 83(4), 647 - 655.
Ribeiro, M. M. B., Pinto, A., Pinto, M., Heras, M., et al. (2011). Inhibition of nociceptive responses after systemic administration of amidated kyotorphin. British Journal of Pharmacology, 163(5), 964 - 973.
Oliveira, M. C., Gano, L., Santos, I., Correia, J. D. G., et al. (2016). Improvement of the pharmacological properties of amidated kyotorphin by means of iodination. MedChemComm, 7(5), 906 - 913.
Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2005). Imbalance between the expression of NK1 and GABAB receptors in nociceptive spinal neurons during secondary hyperalgesia: A c-Fos study in the monoarthritic rat. Neuroscience, 132(4), 905 - 916.

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