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Journal Article
Morgado, C., Pereira-Terra, P., & Tavares, I. (2011). α-Lipoic acid normalizes nociceptive neuronal activity at the spinal cord of diabetic rats. Diabetes, Obesity and Metabolism, 13(8), 736 - 741.
Martins, D., Silva, M., & Tavares, I. (2017). TRPV1 in pain control from the brain. Oncotarget, 8(10), 16101 - 16102.
Ribeiro, M. M. B., Santos, S. S., Sousa, D. S. C., Oliveira, M., et al. (2013). Side-effects of analgesic kyotorphin derivatives: Advantages over clinical opioid drugs. Amino Acids, 45(1), 171 - 178.
Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2006). Secondary hyperalgesia in the monoarthritic rat is mediated by GABAB and NK1 receptors of spinal dorsal horn neurons: A behavior and c-fos study. Neuroscience, 141(4), 2087 - 2095.
Martins, I., Cabral, L., Pinto, A., Wilson, S. P., et al. (2011). Reversal of inflammatory pain by HSV-1-mediated overexpression of enkephalin in the caudal ventrolateral medulla. European Journal of Pain, 15(10), 1008 - 1014.
Martins, I., & Tavares, I. (2017). Reticular formation and pain: The past and the future. Frontiers in Neuroanatomy, 11.
Silva, M., Amorim, D., Almeida, A., Tavares, I., et al. (2013). Pronociceptive changes in the activity of rostroventromedial medulla (RVM) pain modulatory cells in the streptozotocin-diabetic rat. Brain Research Bulletin, 96, 39 - 44.
Pinto, M., Sousa, M., Lima, D., & Tavares, I. (2008). Participation of μ-opioid, GABAB, and NK1 receptors of major pain control medullary areas in pathways targeting the rat spinal cord: Implications for descending modulation of nociceptive transmission. Journal of Comparative Neurology, 510(2), 175 - 187.
Silva, M., Costa-Pereira, J. T., Martins, D., & Tavares, I. (2016). Pain modulation from the brain during diabetic neuropathy: Uncovering the role of the rostroventromedial medulla. Neurobiology of Disease, 96, 346 - 356.
Pinto, M., Castro, A. R., Tshudy, F., Wilson, S. P., et al. (2008). Opioids modulate pain facilitation from the dorsal reticular nucleus. Molecular and Cellular Neuroscience, 39(4), 508 - 518.
Martins, I., De Vries, M. G., Teixeira-Pinto, A., Fadel, J., et al. (2013). Noradrenaline increases pain facilitation from the brain during inflammatory pain. Neuropharmacology, 71, 299 - 307.
Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2004). Nociceptive spinal neurons expressing NK1 and GABAB receptors are located in lamina I. Brain Research, 1003(1-2), 77 - 85.
Oliveira, M. - M., Akerman, S., Tavares, I., & Goadsby, P. J. (2016). Neuropeptide y inhibits the trigeminovascular pathway through NPY Y1 receptor: Implications for migraine. Pain, 157(8), 1666 - 1673.
Morgado, C., Terra, P. P., & Tavares, I. (2010). Neuronal hyperactivity at the spinal cord and periaqueductal grey during painful diabetic neuropathy: Effects of gabapentin. European Journal of Pain, 14(7), 693 - 699.
Pinto, M., Lima, D., & Tavares, I. (2007). Neuronal activation at the spinal cord and medullary pain control centers after joint stimulation: A c-fos study in acute and chronic articular inflammation. Neuroscience, 147(4), 1076 - 1089.
Martins-Oliveira, M., Akerman, S., Holland, P. R., Hoffmann, J. R., et al. (2017). Neuroendocrine signaling modulates specific neural networks relevant to migraine. Neurobiology of Disease, 101, 16 - 26.
Tavares, I. (2004). The molecular metamorphosis of embryology: Implications for medical education. Medical Education, 38(5), 544.
Morgado, C., Pereira-Terra, P., Cruz, C. D., & Tavares, I. (2011). Minocycline completely reverses mechanical hyperalgesia in diabetic rats through microglia-induced changes in the expression of the potassium chloride co-transporter 2 (KCC2) at the spinal cord. Diabetes, Obesity and Metabolism, 13(2), 150 - 159.
Marques-Lopes, J., Pinto, M., Pinho, D., Morato, M., et al. (2009). Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia. Neuroscience, 158(4), 1301 - 1310.
Almeida, A., Leite-Almeida, H., & Tavares, I. (2006). Medullary control of nociceptive transmission: Reciprocal dual communication with the spinal cord. Drug Discovery Today: Disease Mechanisms, 3(3), 305 - 312.
Pinho, D., Sousa, T., Morato, M., Tavares, I., & Albino-Teixeira, A. (2006). Lesion of the caudal ventrolateral medulla prevents the induction of hypertension by adenosine receptor blockade in rats. Brain Research, 1073-1074(1), 374 - 382.
Morato, M., Pinho, D., Sousa, T., Tavares, I., & Albino-Teixeira, A. (2006). Inhibition of nociceptive responses of spinal cord neurones during hypertension involves the spinal GABAergic system and a pain modulatory center located at the caudal ventrolateral medulla. Journal of Neuroscience Research, 83(4), 647 - 655.
Ribeiro, M. M. B., Pinto, A., Pinto, M., Heras, M., et al. (2011). Inhibition of nociceptive responses after systemic administration of amidated kyotorphin. British Journal of Pharmacology, 163(5), 964 - 973.
Oliveira, M. C., Gano, L., Santos, I., Correia, J. D. G., et al. (2016). Improvement of the pharmacological properties of amidated kyotorphin by means of iodination. MedChemComm, 7(5), 906 - 913.
Castro, A. R., Pinto, M., Lima, D., & Tavares, I. (2005). Imbalance between the expression of NK1 and GABAB receptors in nociceptive spinal neurons during secondary hyperalgesia: A c-Fos study in the monoarthritic rat. Neuroscience, 132(4), 905 - 916.

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