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D
Sousa, M. M., Ferraõ, J., Fernandes, R., Guimarães, A., et al. (2004). Deposition and passage of transthyretin through blood-nerve barrier in recipients of familial amyloid polyneuropathy livers. Laboratory Investigation, 84(7), 865 - 873.
Rocha, S., Cardoso, I., Börner, H., Pereira, M. C., et al. (2009). Design and biological activity of β-sheet breaker peptide conjugates. Biochemical and Biophysical Research Communications, 380(2), 397 - 401.
Ferreira, N., Santos, S. A. O., Domingues, M. R. M., Saraiva, M. J., & Almeida, M. R. (2013). Dietary curcumin counteracts extracellular transthyretin deposition: Insights on the mechanism of amyloid inhibition. Biochimica et Biophysica Acta - Molecular Basis of Disease, 1832(1), 39 - 45.
Gonçalves, N. P., Moreira, J., Martins, D., Vieira, P., et al. (2017). Differential expression of Cathepsin E in transthyretin amyloidosis: From neuropathology to the immune system. Journal of Neuroinflammation, 14(1).
Pires, R. H., Karsai, Á., Saraiva, M. J., Damas, A. M., & Kellermayer, M. S. Z. (2012). Distinct Annular Oligomers Captured along the Assembly and Disassembly Pathways of Transthyretin Amyloid Protofibrils. PLoS ONE, 7(9).
Cardoso, I., & Saraiva, M. J. (2006). Doxycycline disrupts transthyretin amyloid: Evidence from studies in a FAP transgenic mice model. FASEB Journal, 20(2), 234 - 239.
Obici, L., Cortese, A., Lozza, A., Lucchetti, J., et al. (2012). Doxycycline plus tauroursodeoxycholic acid for transthyretin amyloidosis: A phase II study. Amyloid, 19(SUPPL. 1), 34 - 36.
Teixeira, C., Costelha, S., Martins, H. S., Teixeira, A., & Saraiva, M. J. (2017). Doxycycline-tauroursodeoxycholic acid treatment: effects in the heart of a transthyretin V30M transgenic mouse model. Amyloid, 24, 80.
E
Gonçalves, P., Martins, H., Costelha, S., Maia, L. F., & Saraiva, M. J. (2016). Efficiency of silencing RNA for removal of transthyretin V30M in a TTR leptomeningeal animal model. Amyloid, 23(4), 249 - 253.
Gonçalves, P., Martins, H., Costelha, S., & Saraiva, M. J. (2017). Efficiency of siRNA for removal of transthyretin V30M in a TTR leptomeningeal animal model. Amyloid, 24, 38 - 39.
Teixeira, P. F., Cerca, F., Santos, S. D., & Saraiva, M. J. (2006). Endoplasmic reticulum stress associated with extracellular aggregates: Evidence from transthyretin deposition in familial amyloid polyneuropathy. Journal of Biological Chemistry, 281(31), 21998 - 22003.
Santos, S. D., & Saraiva, M. J. (2004). Enlarged ventricles, astrogliosis and neurodegeneration in heat shock factor 1 null mouse brain. Neuroscience, 126(3), 657 - 663.
Ferreira, N., Saraiva, M. J., & Almeida, M. R. (2012). Epigallocatechin-3-gallate as a potential therapeutic drug for TTR-related amyloidosis: "In vivo" evidence from FAP mice models. PLoS ONE, 7(1).
Vieira, M., Leal, S. S., Gomes, C. M., & Saraiva, M. J. (2016). Evidence for synergistic action of transthyretin and IGF-I over the IGF-I receptor. Biochimica et Biophysica Acta - Molecular Basis of Disease, 1862(4), 797 - 804.
Cardoso, I., Brito, M., & Saraiva, M. J. (2008). Extracellular matrix markers for disease progression and follow-up of therapies in familial amyloid polyneuropathy V30M TTR-related. Disease Markers, 25(1), 37 - 47.
F
Saraiva, M. J., Sousa, M. M., Cardoso, I., & Fernandes, R. (2004). Familial amyloidotic polyneuropathy: Protein aggregation in the peripheral nervous system. Journal of Molecular Neuroscience, 23(1-2), 35 - 40.
Misumi, Y., Ando, Y., Gonçalves, N. P., & Saraiva, M. J. (2013). Fibroblasts endocytose and degrade transthyretin aggregates in transthyretin-related amyloidosis. Laboratory Investigation, 93(8), 911 - 920.
Pires, R. H., Saraiva, M. J., Damas, A. M., & Kellermayer, M. S. Z. (2017). Force spectroscopy reveals the presence of structurally modified dimers in transthyretin amyloid annular oligomers. Journal of Molecular Recognition, 30(3).
G
Plissonneau, M., Pansieri, J., Heinrich-Balard, L., Morfin, J. - F., et al. (2016). Gd-nanoparticles functionalization with specific peptides for ß-amyloid plaques targeting. Journal of Nanobiotechnology, 14(1).
Oliveira, S. M., Ribeiro, C. A., Cardoso, I., & Saraiva, M. J. (2011). Gender-dependent transthyretin modulation of brain amyloid-β Levels: Evidence from a mouse model of alzheimer's disease. Journal of Alzheimer's Disease, 27(2), 429 - 439.
Altland, K., Benson, M. D., Costello, C. E., Ferlini, A., et al. (2007). Genetic microheterogeneity of human transthyretin detected by IEF. Electrophoresis, 28(12), 2053 - 2064.
H
Soares, M. L., Coelho, T., Sousa, A., Holmgren, G., et al. (2004). Haplotypes and DNA sequence variation within and surrounding the transthyretin gene: Genotype-phenotype correlations in familial amyloid polyneuropathy (V30M) in Portugal and Sweden. European Journal of Human Genetics, 12(3), 225 - 237.
Magalhães, J., & Saraiva, M. J. (2012). The heat shock response in FAP: The role of the extracellular chaperone clusterin. Amyloid, 19(SUPPL. 1), 3 - 4.
Santos, S. D., Fernandes, R., & Saraiva, M. J. (2010). The heat shock response modulates transthyretin deposition in the peripheral and autonomic nervous systems. Neurobiology of Aging, 31(2), 280 - 289.
Noborna, F., O'Callaghan, P., Hermansson, E., Zhang, X., et al. (2011). Heparan sulfate/heparin promotes transthyretin fibrillization through selective binding to a basic motif in the protein. Proceedings of the National Academy of Sciences of the United States of America, 108(14), 5584 - 5589.

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