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T
Fleming, C. E., Mar, F. M., Franquinho, F., Saraiva, M. J., & Sousa, M. M. (2009). Transthyretin internalization by sensory neurons is megalin mediated and necessary for its neuritogenic activity. Journal of Neuroscience, 29(10), 3220 - 3232.
Liz, M. A., Leite, S. C., Juliano, L., Saraiva, M. J., et al. (2012). Transthyretin is a metallopeptidase with an inducible active site. Biochemical Journal, 443(3), 769 - 778.
Sousa, M. M., & Saraiva, M. J. (2008). Transthyretin is not expressed by dorsal root ganglia cells. Experimental Neurology, 214(2), 362 - 365.
Sousa, J. C., Morreale de Escobar, G., Oliveira, P., Saraiva, M. J., & Palha, J. A. (2005). Transthyretin is not necessary for thyroid hormone metabolism in conditions of increased hormone demand. Journal of Endocrinology, 187(2), 257 - 266.
Nunes, A. F., Saraiva, M. J., & Sousa, M. M. (2006). Transthyretin knockouts are a new mouse model for increased neuropeptide Y. FASEB Journal, 20(1), 166 - 168.
Ruano, D., Macedo, A., Soares, M. J., Valente, J., et al. (2007). Transthyretin: No association between serum levels or gene variants and schizophrenia. Journal of Psychiatric Research, 41(8), 667 - 672.
Alemi, M., Gaiteiro, C., Ribeiro, C. A., Santos, L. M., et al. (2016). Transthyretin participates in beta-amyloid transport from the brain to the liver- involvement of the low-density lipoprotein receptor-related protein 1?. Scientific Reports, 6.
Costa, R., Ferreira-Da-Silva, F., Saraiva, M. J., & Cardoso, I. (2008). Transthyretin protects against A-beta peptide toxicity by proteolytic cleavage of the peptide: A mechanism sensitive to the kunitz protease inhibitor. PLoS ONE, 3(8).
Gomes, J. R., Nogueira, R. S., Vieira, M., Santos, S. D., et al. (2016). Transthyretin provides trophic support via megalin by promoting neurite outgrowth and neuroprotection in cerebral ischemia. Cell Death and Differentiation, 23(11), 1749 - 1764.
Vieira, M., & Saraiva, M. J. (2013). Transthyretin regulates hippocampal 14-3-3ζ protein levels. FEBS Letters, 587(10), 1482 - 1488.
Ribeiro, C. A., Oliveira, S. M., Guido, L. F., Magalhães, A., et al. (2014). Transthyretin stabilization by iododiflunisal promotes amyloid-β peptide clearance, decreases its deposition, and ameliorates cognitive deficits in an Alzheimer's disease mouse model. Journal of Alzheimer's Disease, 39(2), 357 - 370.
U
Sousa, M. M., Do Amaral, J. B., Guimarães, A., & Saraiva, M. J. (2005). Up-regulation of the extracellular matrix remodeling genes, biglycan, neutrophil gelatinase-associated lipocalin, and matrix metalloproteinase-9 in familial amyloid polyneuropathy. FASEB Journal, 19(1), 124 - 126.
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Neto-Silva, R. M., Macedo-Ribeiro, S., Pereira, P. J. B., Coll, M., et al. (2005). X-ray crystallographic studies of two transthyretin variants: Further insights into amyloidogenesis. Acta Crystallographica Section D: Biological Crystallography, 61(3), 333 - 339.
Α
Magalhães, J., Santos, S. D., & Saraiva, M. J. (2010). αB-crystallin (HspB5) in familial amyloidotic polyneuropathy. International Journal of Experimental Pathology, 91(6), 515 - 521.

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